Because of a lapse in government funding, the information on this
website may not be up to date, transactions submitted via the
website may not be processed, and the agency may not be able to
respond to inquiries until appropriations are enacted. The NIH
Clinical Center (the research hospital of NIH) is open. For more
details about its operating status, please visit
cc.nih.gov. Updates
regarding government operating status and resumption of normal
operations can be found at
opm.gov.
Glycogen Storage Disease Type VIII MeSH Descriptor Data 2025
An x-linked recessive hepatic glycogen storage disease resulting from lack of expression of phosphorylase-b-kinase activity. Symptoms are relatively mild; hepatomegaly, increased liver glycogen, and decreased leukocyte phosphorylase are present. Liver shrinkage occurs in response to glucagon.
Entry Version
GLYCOGEN STORAGE DIS VIII
Entry Term(s)
Glycogenosis 8
Previous Indexing
Glycogenosis (1966-1974)
Public MeSH Note
91; was see under GLYCOGEN STORAGE DISEASE 1989-90; was GLYCOGENOSIS 8 see under GLYCOGENOSIS 1975-88
Online Note
use GLYCOGEN STORAGE DISEASE TYPE VIII to search GLYCOGENOSIS 8 1975-88
History Note
91(89); was see under GLYCOGEN STORAGE DISEASE 1989-90; was GLYCOGENOSIS 8 see under GLYCOGENOSIS 1975-88
An x-linked recessive hepatic glycogen storage disease resulting from lack of expression of phosphorylase-b-kinase activity. Symptoms are relatively mild; hepatomegaly, increased liver glycogen, and decreased leukocyte phosphorylase are present. Liver shrinkage occurs in response to glucagon.